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Another molecular mechanism for brain damage after I/R concerns matrix metalloproteinases (MMPs) and their ability to interrupt endothelial junctions after restoration of blood flow (Rosenberg and Yang, 2007)

Arabidopsis RING E3 ubiquitin ligase AtATL80 is negatively involved in phosphate mobilization and cold stress response in sufficient phosphate growth conditions

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As expected, markers of apoptosis and an increase in the intracellular calcium concentration were found in blood cells from BD patients compared to healthy individuals (Perova et al., 2008

Significantly decreased mitochondrial membrane potential of cells as a major consequence of AFB1 exposure has been shown in several in vitro and animal studies [137,138]