glutathione in breast milk copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of protein and lipid bovine mammary epithelial cells The physiological role of copper:
Description
The copper peptide may occupy binding sites that would otherwise receive pro-apoptotic signals, effectively blocking some of the damage that DHT causes indirectly
doi: 10.1016/0003-2697(90)90208-Q
P.Mc EwenA
Sanchez-Fayos, P
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