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Membranal expression of calreticulin induced by unfolded protein response in melanocytes: A mechanism underlying oxidative stress-induced autoimmunity in vitiligo

For instance, it has been demonstrated that MitoQ functions by restoring mitophagy in tubular cells through Nrf2-mediated regulation of PTEN-induced putative kinase 1(PINK) transcription, thereby ameliorating mitochondrial oxidative stress and abnormal mitochondrial dynamics [216]

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doi: 10.1158/0008-5472.CAN-16-0587

This effect is mediated by SIRT1/p66Shc pathway (Sampaio et al., 2016)

Both intermediates belong to the group of amino acids that is common in cells but is not used in the synthesis of proteins