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p21 exerts this anti-apoptotic function through several key mechanisms, predominantly when localized in the cytoplasm

doi: 10.1016/j.micpath.2019.04.042 [DOI] [PubMed] [Google Scholar] Noor I., Sohail H., Sun J., Nawaz M

Abstract Viral infections can trigger increased reactive oxygen species (ROS) production and a reduced antioxidant response in the host, leading to redox stress, inflammation, apoptosis, and ultimately, cell and tissue damage, which contribute to disease development

Anti-GAD65 containing cerebrospinal fluid does not Alter GABAergic transmission

doi: 10.1021/acsptsci.0c00223 9 PetrovaBMaynardAGWangPKanarekN

Much of the current work on this topic has focused on uncovering novel sources of H 2 O 2 in response pathways, examining overarching molecular networks that control oxidative burst events, assessing interactions between various environmental and endogenous factors and H 2 O 2 signaling, and studying gene products that protect cellular components during H 2 O 2 responses