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Proteomic analysis of active multiple sclerosis lesions reveals therapeutic targets

However, upon mitochondrial damage, PINK1 degradation is inhibited, resulting in its accumulation on the OMM

Taken together, COPD patients suffer from exaggerated inflammation, increased oxidative stress, and impaired host defense

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The cells were then washed and resuspended in 1 PBS

A previous study demonstrated that the activation of Slc27a2 by HNF4 facilitated the self-renewal of ISCs 14 and our results showed that the overexpression of Slc27a2 prevented the depletion of ISCs, while the inhibition of Fatp2 abolished the protective effects of CBD on ISCs upon IR damage (Fig