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Metabolomics enrichment indicated that SAC modulates the glutathione metabolism pathway by regulating GSH levels, thereby influencing GPX4 stability and activity

Homozygotes and heterozygotes can be symptomatic, although the disease typically is more severe in persons who are homozygous for the deficiency

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Our findings uncover a direct mechanistic link between MYCN-driven ER stress and NEPC progression and highlight NO supplementation as a potential therapeutic strategy to counteract lineage plasticity and metabolic adaptations in NEPC