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Such variability helps to explain the complexity of L-carnitines mechanisms of action and its inconsistent effects across cardiovascular endpoints

[DOI] [PubMed] [Google Scholar] 406.Bellentani S, Pozzato G, Saccoccio G, Crovatto M, Croce LS, Mazzoran L, et al

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(PubMed) Wang Z, Zhu W, Xing Y, Jia J, Tang Y

Although the literature data on the anti-atherogenic activities of CRP is conflicting, some studies conclude that when bound to platelets, native CRP can possibly eliminate the effects of physiological platelet agonists, inhibit platelet secretion (both platelet dense bodies and alpha granule constituents), reduce platelet aggregation, and potentially boost the anti-platelet effect of acetylsalicylic acid [26]

Normal carnitine levels in patients with chronic fatigue syndrome