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however, previous studies done on liver fibrosis and cardiac fibrosis in vivo and in vitro demonstrated that VCN was able to reduce the accumulation of fibrotic components possibly through its main pharmacological mechanism by targeting the PDE1A, where the latter has been identified as a key regulator of fibroblast activation and ECM remodeling in the heart 87

What is their fate in the human body

[DOI] [PubMed] [Google Scholar] 11.Wang M., Zhu P., Jiang C., Ma L., Zhang Z., Zeng X

Franasiak JM, Forman EJ, Hong KH, Werner MD, Upham KM, Treff NR, et al

Received: Revised: Accepted: Published: Version of record: Issue date: DOI:

The mechanism of two-phase motility in the spirochete leptospira: swimming and crawling
