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We next investigated whether the N-terminal or C-terminal domain of AcvB is responsible for this interaction

Hyperoxia-induced reactive oxygen species formation in pulmonary capillary endothelial cells in situ

Roh JH, Lee S, Yoon JH

It is not surprised since LPS-induced cPKC activation has been demonstrated in the recent reports (30, 69)

3.Sikiric

p 0.05) cholesterol (~ 22.51%
