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Moreover, as previously discussed, in a cecal ligation and puncture (CLP)-induced polymicrobial sepsis model, the conditional depletion of GPX4 triggers excessive lipid peroxidation and aggravates systemic inflammatory response that reportedly rely on GSDMD-dependent pyroptosis rather than ferroptosis [12]

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EA is active against human tumor cells in particular through its inhibitory activity on GSTP1-1 by covalent binding of the GSH-EA complex

doi: 10.1104/pp.007062