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However, improving the bioavailability of EGCG via nano-formulation might contribute to a more productive treatment of DM metabolic consequences and vascular complications [196]
Therapeutic inhibition of IGF-1R or downstream AKT/ERK signalingcould therefore potentiate immune checkpoint therapy by restoring MHC-I presentation and reducing PD-L1 expression
Critical role of PUFAs as substrates PUFAs, especially arachidonic acid and adrenic acid, are the primary substrates for lipid peroxidation during ferroptosis
Clin Immunol (2013) 149(3):55665
