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Peng Y, Zhang R, Yang X, et al

Subject of Research : Glutathione transport regulation and redox homeostasis in the endoplasmic reticulum Article Title : SLC33A1 exports oxidized glutathione to maintain endoplasmic reticulum redox homeostasis News Publication Date : April 17, 2026 Web References : 10.1038/s41556-026-01922-y Image Credits : Lori Chertoff / The Rockefeller University Keywords : Antioxidants, Endoplasmic reticulum, Protein folding, Redox homeostasis, Glutathione transport, SLC33A1, Neurodegeneration, Cancer, Mitochondria, Cellular metabolism, Molecular transporters Tags: cancer and protein foldingcellular redox regulationdisulfide bond formation in proteinsendoplasmic reticulum redox homeostasisER protein maturation and traffickingglutathione role in protein foldingliposome-based assay for protein studyneurodegenerative disorder mechanismsoxidative environment in ERoxidized glutathione (GSSG) transportprotein misfolding diseasesSLC33A1 transporter function

R.GutirrezC.et al

The ATPase domain of BAMS and FSHD2 In FSHD2, specific SMCHD1 mutations have been described across the whole coding sequence [59], whereas mutations inducing BAMS are localized within exons 3 to 13 [66]

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Education Medical School: Philadelphia College of Osteopathic Medicine Internship: Tripler Army Medical Center Residency: Tripler Army Medical Center Board Certifications Obstetrics & Gynecology, American Board of Obstetrics & Gynecology Urogynecology and Reconstructive Pelvic Surgery, American Board of Obstetrics & Gynencology Conditions Treated Incontinence Overactive Bladder Nighttime Urination (Nocturia) Vaginal and Pelvic Prolapse Urinary Incontinence Procedures Performed Hysterectomy Sacral Colpopexy Sling Surgery