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Conclusion This study demonstrates that serum CK is associated with skeletal muscle mass in male patients with PC

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204 Similarly, administration of the PRMT5 inhibitor PJ-68 in a CML mouse model with high expression of PRMT5 LSCs could deplete DVL3, thereby inhibiting Wnt/-catenin signaling and significantly prolonging the survival time of a retroviral BCR-ABL-driven CML mouse model

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Primer sequences are presented in Appendix Tables S4S6