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Using cell death kinetic analysis, gene-editing and metabolic tracing, we show that inhibition of RNR-dependent nucleotide metabolism allows for conservation of intracellular GSH and a reduction in ferroptosis susceptibility in cancer cells
An excess of ROS can activate various transcription factors linked to inflammatory responses, exacerbating inflammation
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HongXSongRSongHZhengTWangJLiangYet al
[DOI] [PMC free article] [PubMed] [Google Scholar] 30.Zhang J., Lei H., Hu X., Dong W
(2022) 55:527541.e5
