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In vitro studies, over-expression of the IGF1R has been identified as a typical hallmark of many types of tumors (Werner et al., 2012)

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The current model suggests that a gradient of proteincopper binding affinities and, presumably, relative protein abundance govern the partitioning of copper between cytosolic proteins 50

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Epigenetic remodelling, such as elevated SET domain-containing 7 expression and increased H3 lysine 4 monomethylation at proinflammatory gene promoters (e.g., NF-B p65), sustains monocyte proinflammatory programming [51]