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doi: 10.1016/j.cellimm.2014.02.003 157 MuriJKopfM

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The role of this receptor in mediating zincs influence on TJ protein expression was demonstrated in Gpr39 knockout mice where reduced levels of occludin and ZO-1 were found when compared to wild-type mice (Cohen et al

Frataxin-knockout mouse myoblasts, heart, and skin fibroblasts all showed characteristics of ferroptosis, and Nrf2 was downregulated in Friedreichs Ataxia patients and frataxin-knockout mice, while Nrf2 activators EPI-743 or sulforaphane regulated redox imbalances and rescued ferroptosis in frataxin knockout cells