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The metabolic shifts induced by GLP-1 and GIP activation create conditions where the liver preferentially mobilizes stored triglycerides

Currently, excessive NNMT upregulation is implicated in several scenarios, including obesity, metabolic diseases, muscle cell degeneration, and even some types of cancer [2, 4]

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LIHC is characterized by a series of metabolic alterations such as increased glucose uptake and lactate production [91, 92], upregulated de novo lipogenesis and NADPH production [93, 94], accelerated glutamine consumption [95], elevated amino acids metabolism, and GSH synthesis [9699], as well as attenuated TCA cycles and fatty acid -oxidation [93, 100, 101] when compared with the non-cancerous sample tissues [102]

It has been reported that CD38 low NK-cells exert a higher tumoricidal response compared to CD38 high NK-cells