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Ferroptosis regulation within mitochondria Compelling evidence supports the importance of mitochondria in driving CDI ferroptosis, hence it is reasonable to assume that there might be mitochondria-localized defense systems to prevent ferroptosis

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The expression of HO-1 through NRF-2 depends on the bioavailability of free heme groups, which are toxic for the cells

Research Notes: Tissue Environment Models: Incorporated into experiments analyzing cellmatrix interactions

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