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92 In this mechanism, bacterial GSH is an essential cofactor, and mutants that are defective in GSH biosynthesis cannot inactivate fosfomycin, even though they possess the resistance gene

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Likewise with regard to PD pathology, it is reported that oxidative stress promotes -synuclein aggregation in dopaminergic neurons, and that -synuclein further generates intracellular ROS (Xiang et al., 2013)

Given that the D1-like activation of GluN2B and predominant NMDAR contribute to synaptic plasticity, it would be important to investigate whether MPH may inhibit NMDAR plasticity for neurons expressing D1-like receptors