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Research indicates that ether-bonded glycerolipids increase cellular sensitivity to ferroptosis, whereas ether lipids are crucial for the formation of the extracellular meshwork induced by sulfasalazine (104)

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Specifically, infection leads to the hypomethylation of the AVP promoter region in the posterodorsal medial amygdala, which enhances the expression of vasopressin and increases the activation of vasopressinergic neurons when the animal is exposed to cat scent [21]

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