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Anjum et al., suggested a Cu-induced oxidative stress mechanism because some substituted bis(4,4-dimethyl-3-thiosemicarbazone) Cu(II) complexes show antiproliferative activity to a large extent against tumor cells [8]

However, upon exposure to LFUS, all the tested formulations showed sensitivity to LFUS, triggering the release of their loads

The superior antioxidative stress effects of CBMSCCM were attributed to its rich secretome profile, which included trophic factors such as NeuroD1, neuropilin2, neurturin, nidogen1, NrCAM, NT3, NT4, and VEGF as well as matrix metalloproteinases (MMP-1/3)

Simultaneously, the regulatory functions of IL-22 and IL-33, characterized by anti-inflammatory and cardio-reparative effects, respectively, may counteract the pro-inflammatory actions of IL-6 and IL-1 at various stages of the disease

It was also linked to increased expressions of the vasoprotective markers, decreased oxidative stress, and reduced inflammation in PVAT [94]

PMC 5291841