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122, 203 These same investigators found that mutating apoAI Met residues to Val, mutating Tyr residues to phenylalanine (Phe), or chemically methylating Lys to make its less reactive with lipid carbonyls, all failed to protect apoAI from MPO-induced inhibition of cholesterol efflux via ABCA1

The 'Arctic' APP mutation (E693G) causes Alzheimer's disease by enhanced A[beta] protofibril formation

I wouldnt dismiss the Brasky data entirely, but I also wouldnt extrapolate it to every person taking a B12 supplement

W., Walker, B
The autism increase: research needed on the vaccine connection
