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GSH prevents LOsG-induced pancreatic fibrosis via the TGF/SMAD pathway in vivo Considering the crucial role of TGF-1 in organ fibrosis, we examined whether LOsG contributed to TGF-1 expression by detecting TGF-1 protein levels in pancreatic tissues using western blotting
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Understanding these mechanisms can help improve stimulation procedures and treatment regimens, leading to enhanced nerve healing and functional outcomes
In an effort to investigate whether NMN treatment would have an impact on the health of UMNs diseased with TDP-43 pathology, we utilized dissociated motor cortex cultures established from prpTDP-43 A315T -UeGFP mice, in which CSMN are genetically labeled with eGFP expression that is stable and long-lasting, and when in culture retain their CSMN identity and eGFP expression
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doi: 10.1196/annals.1320.019 40 PennistonKLTanumihardjoSA
