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In conclusion, LEAP-2 inhibits the function of GHS-R1a by reducing constitutive activity, displacing ghrelin from binding sites, and minimizing intracellular signaling activated through receptor internalization, and therefore is a specific and potent endogenous GHS-R1a blocker

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13 There are a number of alternative methods for detection of GST fusion proteins

(2006) NADPH oxidases in cardiovascular health and disease

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Compared to dihydrokavain, lower amounts of urinary metabolites kavain were excreted following its administration but both hydroxylated and ring-opened products were generated