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10.3390/ijms14047109 Int

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Histone H3.3 mutations drive pediatric glioblastoma through upregulation of MYCN

PRECISIONS is rooted in a plausible mechanism that links dysregulated innate immunity to pulmonary fibrosis based on pre-clinical models, identification of genes critical in regulating immunity (i.e., TOLLIP ), and pharmaco-genetic studies that suggest a potential benefit of NAC in IPF patients with the TOLLIP TT genotype