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Although the precise mechanisms underlying KDs effects in PD remain incompletely understood, evidence suggests that the diet can modulate glucose utilization in the brain, neurotransmitter metabolism, insulin signaling, oxidative stress, neuroinflammation, and ion channel activity, particularly potassium channels [20]

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1) Fatigue Glucocorticoid resistance impairs feedback inhibition of the HPA axis, leading to its dysfunction or hyperactivity [11]

NE is the major transmitter via 1 adrenoceptors with potentiation by 1 stimulation

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