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As indicated above the elevated NADH resulting from the metabolism of ethanol results in the impairment of hepatic gluconeogenesis from lactate, alanine (and other amino acids), and glycerol
Triptolide induces mitochondrial apoptosis by downregulating caveolin-1 (CAV-1) mRNA and protein expression, resulting in the activation of the AKT/Bcl-2 signalling axis
TIGAR improve the survival rate of glioma cells under hypoxia
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Mitochondrial and inflammatory changes in sporadic inclusion body myositis
[PubMed] [Google Scholar] 53.Boo YC
