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A Fenton reaction at the endoplasmic reticulum is involved in the redox control of hypoxia-inducible gene expression

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Therefore, it is speculated that ferroptosis may exert its pathogenic effect in SLE by excessive inflammation, which enhances immune response, leading to organ damage and clinical manifestations

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Although CD44 is not involved in paracetamol pharmacokinetics, it is implicated in the inflammatory response to liver injury [43]
