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Description
Similarly, ATMs in lean subjects are supported by fatty acid oxidation (FAO), glycolysis and glutaminolysis (anti-inflammatory macrophage), while ATMs in obese subjects depend more on glycolysis (pro-inflammatory macrophage) [9]
coli strain NCTC 13846 with an MCR-1-mediated colistin resistance
2023;6(6):e2316182
A.ManningJ
June 9-12, 1999
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