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In AD pathology, abnormally modified Tau proteins regulate microglial activation: Hyperphosphorylated Tau enters microglia via CX3CR1 receptor-mediated endocytosis, activating Syk tyrosine kinase signaling to induce phagolysosomal dysfunction and pro-inflammatory mediator release (36,37)

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BPC-157 (Body Protection Compound-157) er et syntetisk peptid som har ftt stor oppmerksomhet i fag- og brukermiljer

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