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The beta-like-globin gene domain in human erythroid cells

B cells promote inflammation in obesity and type 2 diabetes through regulation of T-cell function and an inflammatory cytokine profile

182 Recently, a Cochrane review tried to draw conclusions from 10 studies with a total of 1,450 participants existing data 183 and made the following points: All studies described the presence of PAEC in 41%78.8% of all patients

PMID 15994383

A protection mechanism against excess nitric oxide

Nevertheless, experimental data suggest that the altered intracellular redox status associated with hyperhomo-cysteinemia-induced oxidant stress is the major molecular mechanism responsible for the atherothrombogenicity of moderately elevated circulating homocysteine