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Size: 10 mL

Injections bypass this limitation entirely

Overall, multiple mechanisms including inflammation and reduced FA re-esterification likely explain the impaired buffering capacity in adipocytes during obesity that leads to the spillover of FAs, buildup of toxic lipid metabolites and allosteric regulators that suppress insulin action and increased hepatic glucose output, respectively (Figure 5)

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A study published in Endocrinology demonstrated that chronic treatment with the fragment in obese mice resulted in a reduction of adipose tissue mass without changes in lean body mass or food consumption