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After exposure to oxidative stress (i.e., following hypoxia or inflammation), KEAP1 undergoes a conformational alteration, which prevents it from binding to Nrf2 and leads to the release, accumulation, and translocation of Nrf2 into the nucleus [1, 91] (Fig

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[DOI] [PMC free article] [PubMed] [Google Scholar] Hoshino M, Maeda M, Konishi R, Seki H, Ford PC (1996)

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