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[DOI] [Google Scholar] 86.El-Damarawi MA, Salama ME

During acute ischemia, synaptic vesicles release excessive Zn 2+ that synergizes with glutamate to activate post-synaptic N-methyl-D-aspartate (NMDA) receptors (NMDARs), Ca 2+ -permeable -amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptors (GluR2-lacking), and voltage-sensitive calcium channels (VSCCs), creating a self-perpetuating cycle of cation influx (121123)

Even though estrogen imbalance isnt a part of the diagnostic criteria, we see it a lot clinically

Hirvonen J, Goodwin RS, Li CT, Terry GE, Zoghbi SS, Morse C, Pike VW, Volkow ND, Huestis MA, Innis RB

In contrast, endogenous 4-1BB signaling upregulates PGC-1 through stimulation of the p38-MAPK pathway, resulting in mitochondrial fusion and biogenesis and substantially increasing T cell respiratory capacity