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1.3 Rationale for focusing on C/EBP-inflammasome axis: bridging transcriptional regulation and chronic inflammation in NDs The C/EBP-inflammasome axis has emerged as a critical pathway linking transcriptional regulation to chronic inflammation in NDs ( Table 1 )

Pharmacological strategies that block PERK, NF-B, or NLRP3 have reduced TMAO induced inflammation in preclinical models, indicating their translatable potential

Transcriptional repression of PGC-1alpha by mutant huntingtin leads to mitochondrial dysfunction and neurodegeneration

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[DOI] [PubMed] [Google Scholar] Kovensky J