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Studies in obese murine models showed the peptide restored repressed -AR mRNA levels to levels comparable with lean animals [1]
About this article Cite this article Scheich, C., Sievert, V
Rubino et al., 1998), supporting the idea these symptoms are largely suppressed by this continued post-withdrawal receptor activation
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This has been demonstrated in a particular mouse model with breast carcinoma cells, or with the antibody NZ-1 anti-PDPN for lung metastatization of CHO-expressing human (h)PDPN, or using point-mutated hPDPN-expressing CHO cells
