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Hence, decreased intracellular GSH levels in erlotinib-resistant cells are likely due to the changes in GSH metabolism

The ppar/activator gw 501516 prevents the down-regulation of ampk caused by a high-fat diet in liver and amplifies the pgc-1-lipin 1-ppar pathway leading to increased fatty acid oxidation

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SIRT4 was also found to deacetylate and inhibit mitochondrial malonyl-coenzyme A (malonyl-CoA) decarboxylase (MLYCD) during ad libitum feeding to stimulate mitochondrial fatty acid synthesis, which was prevented by DR (Laurent et al., 2013)

Anabolic signaling was determined as the quantity of pAKT and pRPS6 normalized to Glycerinaldehyde-3-phosphate-Dehydrogenase (GAPDH)

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