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Because of their close relationship in pathophysiology, the management of bleeding from cardiac varices (GOV1s) is similar to that for EV bleeding [177]

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This may be explained by sustained esterification of fatty acids by BMAds even during negative energy balance, in addition to upregulation of PPARy-mediated BMAd adipogenesis

Another mechanism [20] is the stabilization of cell membranes since oxidative stress induces lipid peroxidation, which destabilizes the cell membranes, provoking damage and apoptosis

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