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Despite considerable efforts, using both in vitro and in vivo preparations, the pathophysiological role of anti-GAD antibodies has not yet been clarified, suggesting that other autoantibodies affecting inhibitory neurotransmission might be of importance, because autoimmunity targeting inhibitory synaptic antigens point to a unifying theme of hyperexcitability as the underlying pathomechanism

10.1016/j.indcrop.2012.07.017 6 Amaral de BritoA

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Accordingly, we found that knocking down HSF1 did not compromise acivicin-induced tunicamycin stress resistance (Supplementary Fig

Annals of Neurology

DOI: 10.1038/08398 11