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Similarly, in in vivo experiments, Lip-1, a ferroptosis inhibitor, decreased neuronal death and neuro-inflammation in EBI by protecting mitochondrial functions and reducing lipid peroxidation, thereby ameliorating cerebral edema and neurological deficits after SAH ( 3 The Mechanism of Ferroptosis Studies have indicated that ferroptosis may be induced by different physiological conditions and pathological stress

Treatment of astrocytes with GGC significantly reduced caspase 3 activity (Figure 1B), supporting the protective effects of GGC against oligomeric A 40 -mediated apoptosis

S2CID 24024090

Although these typical changes are not necessarily characteristic of ME/CFS, another autoimmune mechanism may exist in ME/CFS that affects the autonomic control of blood vessel tone and flow autoregulation

The long non-coding RNA MALAT1 activates the NRF2 transcription factor, enhancing the expression of antioxidant genes such as HO1, NQO1, and GCLC, which helps protect cells from oxidative stress-induced damage [167]