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10.2337/dbi19-0014 [DOI] [PubMed] [Google Scholar] 33.Weir GC (2020) Glucolipotoxicity, -cells, and diabetes: the emperor has no clothes

Seleninic or selenol intermediates generated from the oxidation/reduction of DSePA can follow the analogous catalytic steps as discussed for the native GPx enzyme in the earlier section to detoxify the hyperoxides by consuming the thiols [82, 83]

(3) This work also involves the role of ferroptosis-targeting drugs in improving DM and related organs, and critically appraising ferroptosis-targeting drugs for their clinical feasibility beyond glucose control

As a by-product of this adaptation, these cancer cells exhibit increased resistance to genotoxic treatments including ionizing radiation, temozolomide, and cisplatin

Journal of Clinical Endocrinology & Metabolism , 89(7), 3543-3553

[2] [3] [4] [5] [6] [Reaction Scheme 1] [7] enzyme [8] wherein R is an alkyl group (C H , n = 1 to 8)