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circadian rhythm and glutathione An integrative multi-omics analysis leveraging Mendelian randomization subsequent experimental validation prioritizes S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in Circadian rhythm of NRF2 binding

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circadian rhythm and glutathione An integrative multi-omics analysis leveraging Mendelian randomization subsequent experimental validation prioritizes S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in Circadian rhythm of NRF2 binding

PMID 18596934

circadian rhythm and glutathione An integrative multi-omics analysis leveraging Mendelian randomization subsequent experimental validation prioritizes S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in Circadian rhythm of NRF2 binding

J Nat Sci Biol Med

circadian rhythm and glutathione An integrative multi-omics analysis leveraging Mendelian randomization subsequent experimental validation prioritizes S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in Circadian rhythm of NRF2 binding

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circadian rhythm and glutathione An integrative multi-omics analysis leveraging Mendelian randomization subsequent experimental validation prioritizes S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in Circadian rhythm of NRF2 binding

L.Ikeda-SaitoM.SzwedaL

circadian rhythm and glutathione An integrative multi-omics analysis leveraging Mendelian randomization subsequent experimental validation prioritizes S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in Circadian rhythm of NRF2 binding

Cell 148 , 12931307 (2012)

circadian rhythm and glutathione An integrative multi-omics analysis leveraging Mendelian randomization subsequent experimental validation prioritizes S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in Circadian rhythm of NRF2 binding

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