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Although patients with active acromegaly often exhibit increased lean body mass and reduced overall adiposity, their adipose tissue displays an inflammatory molecular phenotype [1, 17].This paradox highlights that metabolic dysfunction in acromegaly is not driven by fat quantity but rather by its altered endocrine function

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Acta Psychiatr Scand 127(2):94114 Cabrero A, Merlos M, Laguna JC, Carrera MV (2003) Down-regulation of acyl-coA oxidase gene expression and increased NF-KappaB activity in etomoxir-induced cardiac hypertrophy

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