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The following experimental details from each study are presented in Table 3: cancer type, chemotherapy agent tested, cell models used (divided by chemosensitive and chemoresistant cell lines), method of MET/HGF blockade employed, observed changes in MET/HGF leading to enhanced MET signalling and the MET-dependent mechanisms of chemotherapy resistance

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Biochemical Analysis Serum enzymes (creatine phosphokinase, CK

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However, in a subsequent 24-week trial involving 536 obese participants, AOD 9604 did not achieve statistically robust, clinically meaningful weight-loss outcomes versus placebo, and the obesity-drug development program was discontinued around 2007