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Engineering those sequence changes back into the original RNA dramatically sped up the production of lab-grown cells infected with hepatitis C

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The loading doses are generally smaller, fractionated injections designed to steadily increase receptor exposure without overwhelming the system in a single large bolus

The C-terminal ligand-binding domain of Clostridium difficile toxin A (TcdA) abrogates TcdA-specific binding to cells and prevents mouse lethality