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Central to this cascade is glutamate-induced hyperactivation of N -methyl- D -aspartate receptors (NMDA receptors), driving pathological calcium influx that overwhelms mitochondrial buffering capacity [103] (Fig

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2006 Mar;11(1):72-6.Acetyl-l-carnitine in the treatment of painful antiretroviral toxic neuropathy in human immunodeficiency virus patients: an open label study

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