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mBio 13, e0041222

Mechanistically, in contrast with the canonical doctrine that GSH globally maintains proteins in a reduced state, we show that GSH depletion selectively oxidizes and promotes the degradation of fatty acid synthase (FASN), a key regulator of lipid metabolism

SLC30A3 responds to glucose- and zinc variations in beta-cells and is critical for insulin production and in vivo glucose-metabolism during beta-cell stress

Store according to institutional laboratory protocols

followed eight patients with congenital lactic acidosis who received oral DCA (5.12 mg/kg every 9 h) for a duration ranging from 5.1 to 7.16 years, during which their renal, liver, electrolyte, and hepatic statuses remained stable [128]

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