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The model predicts that regulation of Met trafficking through 51 binding is an important regulator of downstream Akt and ERK signaling (Fig

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Angiopoietin-2 exacerbates cardiac hypoxia and inflammation after myocardial infarction

We used the Human-GEM model as the metabolic network [16], and mapped the differentially expressed gene (log fold change of genes and the p-value from the differential expression analysis) to the metabolic network using Raven 3.0 [7]