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Nigam SK (2018) The SLC22 transporter family: a paradigm for the impact of drug transporters on metabolic pathways, signaling, and disease
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doi: 10.1146/annurev.micro.55.1.21 65 RitzD.PatelH.DoanB.ZhengM.AslundF.StorzG.et al
The same scenario presents itself during chlorination of the thioesterified piperazate residue in kutznerides by the nonheme Fe(II)-dependent halogenase KthP [26]

Furthermore, an increase in mouse mortality was also observed following an increase in the number of OXA treatments

The amyloid cascade hypothesis which is based on the molecular defects observed in autosomal-dominant EOAD has been severely criticized due to the unsolved paradox: Why do A aggregate into fibrils?, but it is clear that the A sequence, A concentration, and conditions that destabilize A are important factors in aggregation [83, 84]
