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Chac2-mediated GSH metabolic reprogramming drives N1-type polarization of neutrophils To determine whether Chac2 is functionally involved in GSH-driven N1-type polarization, we performed both loss- and gain-of-function experiments using HL60-derived neutrophils

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Specifically, we measured oxygen consumption rate (OCR) to assess mitochondrial respiration, including basal respiration, ATP production, and maximal respiratory capacity

Even for partial tears, proper rehabilitation is essential alongside any peptide protocol, and imaging follow-up helps determine whether conservative management is working

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